Patients receiving treatment for various cancers may experience serious consequences due to the cardiotoxicity of anticancer drugs. The molecular site of action, the immediate and cumulative dose, the mode of administration, the presence of any underlying cardiac conditions, and the demographics of the patient are just a few of the variables that affect how severe this toxicity is. Additionally, treatment with other antineoplastic drugs in the past or present may have had an impact on toxicity. Cardiotoxic side effects may appear right away after drug administration or they may take months or years to become apparent after the patient has had treatment. In this article, we examine some recently approved drugs, as well as some regularly used chemotherapy agents, for their likelihood to produce cardiotoxicity. To more precisely identify which people are at risk for developing cardiotoxicity, more research will be needed. Plans for management and methods to lessen cardiotoxicity must also be established.

Cancer treatment has advanced significantly in recent years, and significant strides have been made in lowering the morbidity and mortality from many types of cancer. A new idea is that cancer can be controlled like diabetes or hypertension through early detection, routine monitoring, and coordinated therapeutic decision-making. Therefore, limiting concomitant conditions is crucial for cancer survivors. The risk of heart disease for many cancer survivors is actually equal to the risk of developing recurrent cancer. Patients with cancer today have access to more sophisticated drug regimens, radiation treatment, and surgical procedures as treatment alternatives. The outcomes for patients are likely to be significantly impacted by several of these treatments, many of which have major potential adverse cardiac effects. Therefore, for these consequences to be effectively managed, recognising them is essential. This review's objective is to highlight drugs used often to treat cancer and their related cardiovascular side effects.

Each chemotherapeutic drug has distinct cardiac effects and the capacity to amplify the negative effects of other drugs. Another significant factor in amplifying damage is radiation therapy. It is crucial to keep in mind that intensively treated cancer patients frequently have severe illnesses, and cause-and-effect correlations are frequently ambiguous. So, using a review of the literature as well as the vast clinical expertise of the Department of Cardiology at The University of Texas M.D. Anderson Cancer Center, we make an effort to summarise the current state of knowledge on the cardiovascular side effects of cancer therapy in this review.